1·There are EGFRs on the membrane of trabecular meshwork cells.
小梁细胞膜上同时存在EGF受体。
2·Conclusion Human trabecular meshwork cells can be successfully cultured in vitro.
结论组织培养方法能够成功体外培养人眼小梁网细胞。
3·The amount of trabecular meshwork pigmentation was not a significant predictor as well.
小梁网的色素沉积程度同样也不是有统计学意义的预后因子。
4·The effect was thought to be mediated via trabecular meshwork glucocorticoid receptors.
这种效应被认为是通过小梁网的糖皮质激素受体介导的。
5·Conclusions Oxidative stress can induce the expression of ELAM-1 in porcine trabecular meshwork cells.
结论氧化应激可诱导猪眼小梁细胞表达ELAM - 1。
6·Trabecular meshwork, ciliary body, retina and optic nerve will be the targets for glaucoma gene therapy.
青光眼基因治疗的靶目标包括眼前节的小梁网和睫状体、眼后节的视网膜神经节细胞等。
7·Histochemical and ultrastructural studies of the trabecular meshwork(TM)of 7 normal human eyes were carried out.
本文对7例正常人眼前房角小梁网进行了组织化学——酸性粘多糖及超微结构的研究。
8·Objective:To evaluate the effect of trabecular meshwork rotated subsclera flap with relaxable suture for glaucoma.
目的：探讨巩膜瓣下小梁组织转位联合巩膜瓣可松解缝线术治疗青光眼的疗效。
9·Glucocorticoid effects on the human trabecular meshwork can be used as a model system in which to study glaucomatous damage to the trabecular meshwork.
糖皮质激素对小梁网的作用可作为一个良好模型用于研究青光眼的发病机制。
10·The apoptosis of retinal ganglion cells and trabecular meshwork cells plays an important role in the pathogenesis, diagnosis and management of glaucoma.
青光眼视网膜神经节细胞及小梁细胞的凋亡在青光眼的发病机制、诊断治疗中发挥重要作用。